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Then the vector control called telemetry function will call the digital control and signal transmitter, The digital control and signal transmitter will control to the desired signal in the desired control point. Then the digital control and signal transmitter will control the work of the switch and automatic gate system, and the switch and automatic gate system will control to the desired switch points. Then all kinds of illumination or cooling will be controlled, and the equipment will start. The question is whether I can tell you what the function will be named?
A:
is it like hydraulic or electromagnetic..
Hydraulics are pumps which are activated by gravity, so the motion is linear, as is the travel of a fluid, and pressure is proportional to speed.
Electromagnetics are, in basic form, switches which are activated by magnets.
If you know basic electronics and have an LCD with a touch screen, play with it a bit and you’ll get it.
Will it be used for controlling a wind turbine?
Yes, or a light show, or a water park, for example.
Long-term prostaglandin E2-receptor antagonists inhibit testicular steroidogenesis in the fetal rat but do not alter developmental differentiation and hormonal responsiveness of the seminiferous tubules.
In view of its documented androgenic activity in the fetus, prostaglandin E2 (PGE2) represents an interesting target for regulatory pathways during perinatal development. Using immunohistochemical methods we investigated the role of PGE2-receptors in the fetal male gonad of rats at day 15 of gestation. Fetal testes were obtained from control females treated with either vehicle or a PGE2-receptor antagonist, AH23848, for 4 days prior to mating and during gestation. An Ah23848 concentration of 25 microg g(-1) body weight/day, was sufficient to inhibit completely long-term the anti-androgenic action of PGE2 in the testis. Immunolocalisation showed, however, that it did not prevent the developmental differentiation of Leydig cell populations. The maturation pattern of the seminiferous tubule was not altered by PGE2-receptor blockade and spermatogenesis was normal in both fetuses from control and AH23848-treated rats at day 15 of gestation. The hormone sensitivity of the fetal seminiferous epithelium was also normal. Our results suggest that the influence of PGE2 on
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